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This is in no way intended to be medical advice or a diagnosis tool. (If you did that you are arguably a tool). I just hope to give a basic run down on diabetes in case you are in the wtf category!
TYPE 1 DIABETES
Diabetes mellitus type 1 (Type 1 diabetes, IDDM, juvenile diabetes) results from autoimmune destruction of insulin producing beta cells of the pancreas. Lack of insulin leads to increased blood and urine glucose. Symptoms are frequent urination, increased thirst, increased hunger, and weight loss.
Type 1 diabetes is fatal unless treated with insulin. Insulin can be adminstered daily via needles or pump. There is no cure.
Most people who develop type 1 are otherwise healthy.
Although the cause of type 1 diabetes is still not fully understood it is believed to be of immunological origin.
Type 1 treatment must be continued indefinitely in all cases. Treatment need not significantly impair normal activities, if sufficient patient training, awareness, appropriate care, discipline in testing and dosing of insulin is taken. However, treatment is burdensome for many people.
Complications may be associated with both low and high blood sugar. Low blood sugar may lead to seizures or episodes of unconsciousness and requires emergency treatment. High blood sugar may lead to increased tiredness and can also result in long term damage to organs.
*http://en.wikipedia.org/wiki/Diabetes_mellitus_type_1
MODY
Maturity onset diabetes of the young.
TYPE 1.5 DIABETES
Also known as Latent Autoimmune Diabetes of Adults (LADA), and describes slow-onset Type 1 autoimmune diabetes in adults.
In Type 1 diabetes, the rate of beta-cell destruction is quite variable, being rapid in some individuals (mainly infants and children) and slow in others (mainly adults) so basically it's type 1 developing in adults. LADA is a genetically-linked hereditary, autoimmune disorder that results in the body mistaking the pancreas as foreign body and responding by attacking and destroying the insulin-producing beta islet cells of the pancreas. Simply stated, autoimmune disorders, including LADA, are an "allergy to self.”
Adults with LADA are frequently initially misdiagnosed as having TYPE 2 based on age, not etiology.
*http://en.wikipedia.org/wiki/Latent_autoimmune_diabetes
TYPE 2 DIABETES
Diabetes mellitus type 2 – formerly non-insulin-dependent diabetes mellitus (NIDDM) or adult-onset diabetes – is a metabolic disorder that is characterized by high blood glucose in the context of insulin resistance and relative insulin deficiency. Diabetes is often initially managed by increasing exercising and dietray modification. As the condition progresses, medications may be needed.
Unlike TYPE 1 there is very little tendency toward ketoacidosis though it is not unheard of. Long-term complications from high blood sugar can include increased risk of heart attack, stroke, amputation and kidney failure.
PRE - DIABETES
Pre-diabetes indicates a condition that occurs when a person's blood glucose levels are higher than normal but not high enough for a diagnosis of type 2 diabetes. Many people destined to develop type 2 diabetes spend many years in a state of pre-diabetes.
*http://en.wikipedia.org/wiki/Type_3_diabetes#Classification
TYPE 3 DIABETES
Im a type 3 !! I dont have diabetes on my physical person, but I have to manage it for my baby son!
NEONATAL DIABETES
Neonatal diabetes mellitus (NDM) is a monogenic form of diabetes that occurs in the first 6 months of life. It is a rare disease, occurring in only one in 100,000 to 500,000 live births. Infants with NDM do not produce enough insulin, leading to an increase in blood glucose NDM can be mistaken for the much more common type 1 diabetes, but type 1 diabetes usually occurs later than the first 6 months of life. In about half of those with NDM, the condition is lifelong and is called permanent neonatal diabetes mellitus (PNDM). In the rest of those with NDM, the condition is transient and disappears during infancy but can reappear later in life; this type of NDM is called transient neonatal diabetes mellitus (TNDM). Specific genes that can cause NDM have been identified.
Symptoms of NDM include thirst, frequent urination and dehydration. NDM can be diagnosed by finding elevated levels of glucose in blood or urine. In severe cases, the deficiency of insulin may cause the body to produce an excess of acid, resulting in a potentially life-threatening condition called ketoacidosisMost fetuses with NDM do not grow well in the womb and newborns are much smaller than those of the same gestational age, a condition called intrauterine growth restriction. After birth, some infants fail to gain weight and grow as rapidly as other infants of the same age and sex. Appropriate therapy improves and may normalize growth and development.
*http://en.wikipedia.org/wiki/Neonatal_diabetes
GESTATIONAL DIABETES
On set and duration during pregnancy only.
